The systemic administration of vasopressin leads to the stimulation of a family of receptors on vascular smooth muscle cells, namely arginine vasopressin receptors (AVPR)1a, 1b, 2, oxytocin receptors, as well as some purinergic receptors. Thus, leading to a catecholamine-independent contraction of arterial blood vessels. This pressor effect is typically small in healthy individuals, but may play a larger role in patients with sepsis.
Conventional RRT in Sepsis vs Furosemide Stress Test Winner: Furosemide Stress Test in AKI 4-3 split The Furosemide stress test is a win for the practical over the possible. [...]
See the Current Standings! | For more on NephMadness 2015 | #NephMadness or #CCRegion on Twitter Critical care was a hotbed of controversy with no knock outs, all of the [...]
Submit your picks! | For more on NephMadness 2015 | #NephMadness or #CCRegion on Twitter The critical care region is packed with interesting story lines, SLED was a bubble [...]